Unit 6: Case Study 1
1. An ulcer starts by eroding the mucosa of the G.I tract wall. What functions of digestion or reabsorption might be lost if this layer is no longer functional? What functions will be compromised if the ulcer eats through the submucosa and then the muscularis?
Absorption would not happen correctly because some of the ingested and secreted may seep out of the lumen. This could also create a pathway of entry for pathogens, if the ulcer ate its way through to the muscularis mucosa and lose some control of defecation.
2. If Zachary has a peptic ulcer affecting his stomach and or duodenum, which components of the peritoneum will be affected?
If the ulcer eats a hole into the wall …show more content…
Inflammation of the small intestine is called gastroenteritis. The symptoms that occur with this disease include diarrhea, abdominal cramps, nausea and vomiting. All of these symptoms can cause a fever and it will last anywhere from 3-7 days.
9. With Zachary’s new diet, which type of lipoprotein will decrease in circulation the most? Which type of lipoprotein do most people wish they could decrease and why? Which one do most people wish they could increase?
VLDLs will decrease with Zachary’s new diet. Most people wish they could decrease LDLs because LDL cholesterol is known as “bad” cholesterol, and people have too few LDL receptors. Their body cells remove LDL from the blood less effectively and develop fatty plague. People wish they could increase HDLs which is known as “good” cholesterol.
10. In a healthy person other molecules can be substituted into the pathways of glucose catabolism when the blood glucose level is low. What specific molecules will Zachary now have problems making during times of low glucose and what is the normal source of molecules?
Zachary will have problems making glycogen, which is mobilized and converted to glucose by gluconeogenesis when the blood glucose concentration is low. Glucose may also